Friday, April 7, 2006, 11:00 am — John Dunn Seminar Room, Bldg. 463
Oxidative stress can lead to damage of cellular components at high exposure levels or trigger a defence network and hence an adaptive response after exposure of plants to low metal concentrations. Cu is an essential micronutrient, but its redox properties contribute to its inherent toxicity, i.e. direct production of reactive oxygen species (ROS). Cd on the other hand is non-essential, not redox-active. Due to the different chemical behaviour of both metals, it is obvious that oxidative stress must be arising from different processes. A. thaliana seedlings were exposed to 1, 5 and 10 µM Cd or Cu during 24h. Focus of the analyses was on 3 different processes contributing to the cellular redox balans: (1) origin of the ROS, (2) nature of the ROS induced and (3) the antioxidative defence network. Furthermore the impact of a combined exposure was investigated in order to retrieve information concerning responses in a realistic multipollution context.
Hosted by: Daniel van der Lelie
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